Preprint Review Version 1 Preserved in Portico This version is not peer-reviewed

Influenza virus infection, interferon response, viral counter-response and apoptosis

Version 1 : Received: 30 May 2017 / Approved: 30 May 2017 / Online: 30 May 2017 (07:13:09 CEST)
Version 2 : Received: 12 August 2017 / Approved: 14 August 2017 / Online: 14 August 2017 (04:41:22 CEST)

A peer-reviewed article of this Preprint also exists.


Human influenza A viruses (IAVs) cause global pandemics and epidemics, which remain serious threats to public health because of the shortage of effective means of control. To combat the surge of viral outbreaks, new treatments are urgently needed. Developing new virus control modalities requires better understanding of virus-host interactions. Here we describe how IAV infection triggers cellular apoptosis, and how this process can be exploited towards development of new therapeutics, which might be more effective than the currently available anti-influenza drugs.


influenza virus; apoptosis; antiviral agent; innate immunity; host response


Biology and Life Sciences, Immunology and Microbiology

Comments (0)

We encourage comments and feedback from a broad range of readers. See criteria for comments and our Diversity statement.

Leave a public comment
Send a private comment to the author(s)
* All users must log in before leaving a comment
Views 0
Downloads 0
Comments 0
Metrics 0

Notify me about updates to this article or when a peer-reviewed version is published.
We use cookies on our website to ensure you get the best experience.
Read more about our cookies here.