Influenza Virus Infection, Interferon Response, Viral Counter-Response, and Apoptosis

Jung Min Shim; Jinhee Kim; Tanel Tenson; Ji-Young Min; Denis E. Kainov

doi:10.20944/preprints201705.0209.v2

Preprint Review Version 2 Preserved in Portico This version is not peer-reviewed

Version 1 : Received: 30 May 2017 / Approved: 30 May 2017 / Online: 30 May 2017 (07:13:09 CEST)
Version 2 : Received: 12 August 2017 / Approved: 14 August 2017 / Online: 14 August 2017 (04:41:22 CEST)

Human influenza A viruses (IAVs) cause global pandemics and epidemics, which remain serious threats to public health because of the shortage of effective means of control. To combat the surge of viral outbreaks, new treatments are urgently needed. Developing new virus control modalities requires better understanding of virus-host interactions. Here we describe how IAV infection triggers cellular apoptosis, and how this process can be exploited towards development of new therapeutics, which might be more effective than the currently available anti-influenza drugs.

influenza virus; apoptosis; antiviral agent; innate immunity; host response

Biology and Life Sciences, Immunology and Microbiology

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Influenza Virus Infection, Interferon Response, Viral Counter-Response, and Apoptosis

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