1. Introduction
Eating disorders (EDs) form a group of syndromes characterized by eating behaviors and psychological disturbances, accompanied by changes in weight and/or social alterations. These disturbances significantly influence social functioning and quality of life [
1,
2]. People with eating disorders are vulnerable to developing severe somatic complications that can cause a high risk of suicide [
3] and elevated mortality rates [
4]. Specific disorders, such as Anorexia Nervosa (AN) can reach mortality rates between 5-20% [
5].
Eating disorders have a high prevalence in the population. It has been reported [
6], over the 2000-2018 period, that the lifetime weighted means of EDs were 8.4% (3.3-18.6%) for women and 2.2% (0.8-6.5%) for men. The weighted mean prevalence of DEs at 12 months were 2.2% (0.8-13.1%) for women and 0.7% (0.3-0.9%) for men. More recently, it is shown [
5] that the lifetime and 12-month joint prevalence of ED was 0.91% (95% CI, 0.48-1.71) and 0.43% (95% CI, 0.18-0.78), respectively. The pooled lifetime and 12-month prevalence, by subgroups and including all types of ED, was 1.69% and 0.72%, respectively. The lifetime prevalence of Anorexia Nervosa (AN), Bulimia Nervosa (BN) and Binge Eating Disorder (BED) was 0.16% (95% CI, 0.06-0.31), 0.63% (95% CI, 0.33-1.02) and 1.53% (95% CI, 1.00-2.17), respectively. The lifetime prevalence of EDs in Western countries was 1.89% and was high at 2.58% in women.
An aspect little studied but relevant to eating behavior, and related to compulsive forms of eating, is emotional eating. In this regard, two eating behaviors can be distin-guished, one based on emotional (irritability, anxiety, or stress) cues (emotional eating) and the other on external (smell or sight of attractive food) cues [
7]. Emotional eating is thus regarded as the tendency to overeat in response to negative emotions, such as anxiety, depression, or irritability [
8,
9]. Has shown to be associated with weight outcomes, both in respect to weight gain over time and difficulties with weight loss and weight loss maintenance.
It has been shown that the stress is related to emotional eating among adolescents, which in turn increases the risk of development and continuation of overweight/obesity [
10]. The results of this research indicated that higher cortisol reactivity, but not subjective stress reactivity, predicted subjective emotional eating, over and above the impact of chronic stress. Neither chronic stress nor subjective or objective stress reactivity predicted objective emotional eating after stress induction. The findings point to the role of chronic stress and cortisol reactivity as risks for greater perceived emotional eating among adolescents, while clarifying the differences between perceived and objective emotional eating.
Emotional eating is a common problem among people struggling with weight issues [
11]. Emotional eaters are particularly prone to consuming foods high in fat, sugars, and calories in response to negative emotions [
12]. These eating habits, combined with higher body weight, place emotional eaters at increased risk for diabetes and heart disease [
13,
14]. Emotional eaters are half as likely as non-emotional eaters to achieve the 10% weight loss goal of standard behavioral weight loss treatment [
15]. It should also be remembered that emotional eating may be the first step toward developing a binge eating disorder and its extreme subtypes, such as food addiction [
16]. In addition, emotional eating is manifest differently in men and women. In men, it has been associated with body mass index and uncontrolled eating; however, in women, emotional eating has been linked to uncontrolled eating, anxiety, and poor sleep quality [
17].
The literature shows that in stressful situations, out of a group of 638 women (18-39 years) 12.4% had high scores in emotional eating and 40.4% could be considered moderate emotional eaters [
18]. Similarly, in Italy, in an equivalent situation, and out of 602 people (18-79 years) about half of the cohort increased their food intake to feel better (55 %) and used food in response to feelings of anxiety (48.7%) [
19].
It should be considered that eating behavior does not only refer to the type of relationship established with the food and the quantity ingested. We have already seen the need to consider the quality of the food ingested. Evaluating the quality of the diet, allows obtaining necessary information of great interest to know the health status of people, where healthy eating is crucial to obtain a good quality of life that lasts through the years, and allows preventing overweight, obesity, and chronic non-communicable diseases in adulthood [
20,
21].
Evaluation instruments have been developed to obtain an index of healthy/unhealthy food intake ([
22], and others) also in Spanish population [
23]. For its development, a quantitative and qualitative evaluation of the diet was taken as a basis, which is compared with recommended intakes in each of the 10 groups or areas that make up the index and that represent aspects of interest in nutrition. Diet quality indices aim to evaluate diet in a general way and categorize individuals according to whether they are more or less compliant with behaviors considered as healthy. Healthy eating includes sufficient intakes of fruits, vegetables, grains, dairy, proteins, nuts, and oils; while limiting saturated fats, salt, and empty calories [
24,
25,
26,
27].
Basically, the data related to diet quality show a very similar pattern among different countries (Argentina, Canada, United States, France, Chile, Turkey, Brazil, etc.). It is indicated that more than half of the adult population had a poor quality diet, observing a less healthy diet in men, young adults and those with lower educational level [
28,
29,
30,
31,
32,
33,
34,
35].
On the other hand, a construct that may help to understand how eating behavior problems develop and are maintained is that of psychological inflexibility. Psychological flexibility refers to the possibility of fully embracing unpleasant private events in the present, without attempting to modify them. Rather, the individual is encouraged to let go of a struggle against discomfort and instead fully feel, engage with, and view these events for what they are as a method of serving one’s own values [
36,
37]. The psychological flexibility model (fundamental to Acceptance and Commitment Therapy; ACT) promotes adaptive coping through six processes of skills-acceptance, cognitive defusion, awareness of the present moment, ability to see oneself in context, values, and committed action [
36,
38,
39].
Psychological inflexibility is strongly and positively associated with various psychopathologies, including anxiety, depression, and eating disorders [
40,
41,
42], while it is negatively associated with quality of life, perceived health, and positive emotional experiences [
36]. In addition, evidence in non-clinical samples suggests that psychological inflexibility and experiential avoidance (the unwillingness to stay in touch and consequently avoid internal states such as unpleasant thoughts, feelings, and sensations) play a key role in the relationship between negative emotional states and the onset of emotional eating [
9,
43,
44].
With respect to eating behaviors, the psychological inflexibility is associated with increased binge eating among individuals who meet the criteria for overweight/obesity [
45]. Moreover, several studies have indicated that cognitive diffusion techniques (cognitive defusion), as a component of psychological flexibility, reduce cravings and objective eating behaviors [
46]. Thus, psychological inflexibility appears to produce barriers that make it difficult to change weight- and eating-related conditions [
47]. Note that no research has been found linking healthy eating patterns and psychological inflexibility.
In this context, the aim of the present research is to determine the relationship between the construct of psychological inflexibility and behaviors related to emotional eating and the healthy eating index. It is expected to find, as a first hypothesis, that women, compared to men, will present a greater psychological inflexibility. The second hypothesis predicts that women will show scores indicating a greater association with emotional eating than men. As a third hypothesis, it is proposed that people with high scores in psychological inflexibility will show a greater association with the presence of emotional eating. Similarly, as a fourth hypothesis, psychological inflexibility is predicted to be negatively associated with healthy dieting.
3. Results
The mean age of the participants was 27.21 years (SD = 10.67). Notably, women were significantly younger than men (
t = 2.108,
p = .035). Specifically, women tend to fall within the 18–24-year age range, whereas men are predominantly in the 25–44-year age group (
Table 1), with an effect size of Cramer’s V = 0.138. Differences in educational level were also found, with more women holding university-level qualifications than men, who tend to have medium levels of education (Cramer’s
V = 0.243). As expected, there are also gender differences in body composition. Men, on average, were heavier and taller than women. Consequently, differences were observed in BMI, with women predominantly falling into the underweight category while a higher number of men were classified as overweight (Cramer’s
V = 0.70).
Table 2 compares the mean scores for each variable according to the gender of the participants. Regarding emotional eating scores, no significant gender differences were found (
Cohen’s d = 0.14). However, it is worth noting that men scored higher than women, suggesting a lower use of food for emotional management. Significant differences were found for F1.- EADES (Self-efficacy in Emotion- and Stress-Related Eating) scores, with men showing higher levels of self-efficacy or reduced reliance on food as a coping mechanism for dealing with negative emotions (
Cohen’s d = 0.34). However, no differences were found in the F2-EADES (Self-confidence in Emotion -and Stress-Related Eating) or in the F3-EADES (Appraisal of Resources and Ability to Cope).
Inspection of the psychological inflexibility scores according to gender revealed that women obtained significantly higher scores, with a medium effect size (Cohen’s d = 0.42). There was a tendency for women to place themselves in the high and medium inflexibility categories, while men placed themselves in the low inflexibility group (Cramer’s V = 0.193). In the case of women, 13.3% were found to have high inflexibility scores, 40.4% medium, and 46.3% low. In contrast, 7.8% of men were classified as having high inflexibility, 25.7% as medium, and 66.5% as low inflexibility.
When examining the scores obtained on diet quality according to the healthy eating index for the Spanish population, women tended to report consuming a healthier diet than men, with a medium effect size (Cohen’s d = 0.25). However, when the participants were grouped according to the categories established for diet quality (healthy, needs changes, unhealthy), we observed that both men and women were grouped similarly, with no significant gender differences (Cramer’s V = 0.082).
When the participants were categorized according to their psychological inflexibility scores (
Table 3), no significant differences were found in the healthy eating index between the three groups. However, group differences were found in the response to emotional eating. As psychological inflexibility decreased, higher scores were obtained in emotional eating, indicating a reduced reliance on food as an emotional regulator. Thus, it was found that for these differences in the emotional eating scale, the effect size was large
(ŋ2 = 0.18). For Factor 1-EADES, the effect size of self-efficacy in emotion- and stress-related eating was medium
(ŋ2 = 0.12). For Factor 2-EADES, self-confidence in emotion- and stress-related eating, the effect size was small
(ŋ2 = 0.03), and for Factor 4-EADES, appraisal of resources and ability to cope, the effect size was large
(ŋ2 = 0.16).
Post hoc comparisons using the Bonferroni test revealed significant differences between the groups in the emotional eating scale, where a < b (p < .001); a < c (p < .001); b < c (p < .001). For the subscale of self-efficacy in emotion- and stress-related eating, differences between all groups were found, with a < b (p < .001); a < c (p < .001); b < c (p < .001). Similarly, the three groups differed significantly on the subscale appraisal of resources and ability to cope, with a < b (p < .001); a < c (p < .001); b < c (p < .001). However, for factor two of the scale, self-confidence in emotion- and stress-related eating, the source of the difference emerges from the low inflexibility, which differs from the other two groups, with a < b (p = .002) a < c (p < .001); b = c (p = .623).
Relationships between the variables were analyzed using Pearson’s correlation test to facilitate stepwise linear regression analysis. The resulting correlations indicate that high scores in psychological inflexibility are negatively related to scores on the emotional eating scale. However, no relationship was found with the healthy eating index (
Table 4).
The regression analysis (
Table 5) was conducted to generate models that can help to explain and predict emotional eating scores. For this purpose, psychological inflexibility, healthy eating index, and BMI were considered as predictor variables. The inclusion of BMI as a predictor variable was motivated by its low but significant correlation with emotional eating and the fact that the existing literature suggests that emotional eating is associated with body weight.
Psychological inflexibility alone as a predictor variable accounts for 21.2% of the variance in emotional eating scores, showing a negative predictive relationship (β = - .461). When the healthy eating index is introduced into the model, the explanatory capacity increases slightly to 23.9%. However, the predictive power of psychological inflexibility for emotional eating is reduced (β = - .455) and the healthy eating index contributes to the model with a negative coefficient (β = - .164). Finally, when BMI is added to the model, its overall explanatory power increases to 24.6%. It should be noted that while BMI makes a significant contribution, the increase in the predictive capacity of the model is small (β = - .082).
Finally, we would like to highlight data that might be considered of particular interest. There were no significant differences in psychological inflexibility or emotional eating based on participants’ education levels. However, differences were observed in the healthy eating index (F(2, 702) = 10.583, p < .001). Specifically, this difference stemmed from the fact that the participants with a university education showed different dietary patterns to those with secondary education (p < .001).
Differences in psychological inflexibility were found according to age (F(2,704) = 9.230, p < .001), where the highest levels of inflexibility were reported among participants aged 18-24 years compared to the 25-44-year age group (p = .009) and the 45-62-year group (p = .001). In contrast, no differences were observed between the two older groups. Similarly, age differences were found in the healthy eating index (F(2,704) = 3.817, p = .022), where the 45-62-year group had a healthier diet than both the 18-24-year (p = .025) and 25-44-year group (p = .033), with no differences between the two younger age groups. Finally, no differences in emotional eating were found according to age group.
No significant differences were found in psychological inflexibility based on BMI categories (F(3,704) = 2.024, p = .109). However, differences are found in emotional eating (F(3,704) = 3.292, p = .020) and its factors F1-EADES (F(3,704) = 4.510, p = .004) and F2-EADES: (F(3,704) = 2.614, p = .050), but not F3--EADES (F(3,704) = 1.086, p = .354). Participants categorized as obese appeared to rely more on food as an emotional regulator than both the underweight group (p = .030) and the normal weight group (p = .029), but not the overweight group (p = .355). No differences were found among the remaining groups. A slight trend was observed regarding the healthy eating index, although with a residual difference (F(3,704) = 2.318, p = .074), with the overweight group tending to have a better-quality diet than the obese group.
Finally, it should be noted that only 11.3% of the participants reported having a healthy diet. In comparison, a significant majority (88.7%) need to make significant changes, and in 17.6% of cases, the diet was characterized as unhealthy and a cause for concern.
4. Discussion
The present research aimed to explore the relationship between the construct of psychological inflexibility and behaviors related to emotional eating and the healthy eating index. Our first hypothesis proposed that women would show higher levels of psychological inflexibility than men. This prediction was supported by our data, revealing that women tend to fall into the high and medium categories of psychological inflexibility. In contrast, men are predominantly categorized as having low inflexibility. These findings align with those reported in the existing literature [
41,
43,
44,
54,
55], highlighting gender-based differences in various disorders, with women typically considered more vulnerable [
56,
57].
The second hypothesis predicted that women would exhibit stronger associations with emotional eating than men. This hypothesis was only partially supported by the data. While the analyses revealed no significant gender differences in the total scale score, there seems to be a tendency for men to obtain higher scores on the emotional eating test. However, gender differences were found in the self-efficacy in emotion- and stress-related eating factor, with men obtaining higher scores than women, indicating that men are less inclined to use food as a coping mechanism for managing emotions.
These results partially support the notion that emotional eating manifests differently in men and women [
17]. It is well-established that women show a greater propensity for emotional eating in response to negative emotions, including feelings of anxiety, anger, frustration, and depression [
55,
58,
59]. Moreover, it is worth noting that, in line with these findings, emotional eating appears to be linked more strongly to increased cortisol than subjective stress reactivity. Interestingly, neither chronic stress nor subjective or objective stress reactivity were shown to be predictors of objective emotional eating in response to stress induction [
10]. Thus, it has been suggested that cortisol hyperreactivity could serve as a marker of stress, particularly among women who develop depression [
60]. Additionally, other potential factors have been proposed to explain why women may be particularly prone to emotional eating, including mood changes related to hormonal variations associated with the menstrual cycle [
61,
62] and emotional changes linked to a heightened cortisol response.
Our third hypothesis proposed that individuals with high scores in psychological inflexibility would show a greater tendency to exhibit emotional eating. Our analyses supported this hypothesis, demonstrating that as psychological inflexibility scores increase, the scores on the emotional eating test decrease. This decrease in scores indicates an increased reliance on food as an emotional regulator. It is also worth noting that differences were found between the three inflexibility groups on the test, both in the total score and for two of the subscales. Specifically, these differences were observed in the subscale of self-efficacy in emotion and stress-related eating and in the subscale of appraisal of resources and ability to cope. However, for factor two of the scale — self-confidence in emotion- and stress-related eating — the low inflexibility group differed from the other two groups.
These results support other findings in the literature suggesting that psychological inflexibility is positively associated with eating behavior problems [
42], showing an inverse relationship with positive emotional experiences [
36]. It has been found that psychological inflexibility has a significant negative predictive capacity for emotional eating either as a single variable in the model (
β = - .461), when the healthy eating index is introduced into the model (
β = - .455) or when BMI is added to these two variables (
β = - .456).
Data from the literature have consistently emphasized the significance of psychological inflexibility and experiential avoidance in understanding the relationships between negative emotional states and the onset of emotional eating [
9,
43,
44]. In this regard, research has demonstrated that psychological inflexibility interventions can potentially mitigate eating-related problems [
46]. One perspective for understanding this situation involves considering that individuals who struggle with emotional regulation may find it challenging to control their impulses in certain emotional situations and may possess limited strategies for managing their emotions effectively [
63]. Consequently, they may resort to coping mechanisms associated with psychological inflexibility in such situations. This can be manifest as attempts to avoid distressing situations or linking a thought to an experience, leading to a reluctance to engage in positive actions, since similar efforts have not yielded the desired outcome in the past [
63]. In essence, individuals who encounter difficulties in managing and regulating emotions tend to exhibit behaviors characteristic of psychological inflexibility [
63], leading to increased symptoms of various forms of psychopathology, particularly anxiety and depression [
64,
65]. Therefore, emotional dysregulation is thought to indirectly impact emotional disorders through its association with psychological inflexibility, given that both emotional dysregulation and psychological inflexibility are related to psychopathology [
66].
Finally, our fourth hypothesis predicted a negative association between psychological inflexibility and a healthy diet. However, our data do not support this hypothesis. It was observed that levels of psychological inflexibility are not correlated with diet quality. The data provided indicate that a substantial percentage (88.7%) of the participants need to make significant dietary changes, while 17.6% fell into the category of having a diet that is worryingly unhealthy. These values align with (albeit slightly higher than) those reported in Spain in the general population by Norte-Navarro and Ortiz-Moncada [
23]. Their study, which included 29.478 individuals [
67], found that 72% of participants required changes in their dietary habits.
The literature has consistently demonstrated a strong relationship between the construct of psychological inflexibility and eating behavior problems [
42], including the use of food as a means to regulate negative emotions [
43]. Importantly, modifying psychological inflexibility can potentially reduce eating problems [
46]. However, no previous studies have explored the relationship between psychological inflexibility and diet quality. The absence of a direct relationship between diet quality and psychological inflexibility may suggest that this construct is more closely linked to the patterns of food consumption and eating behaviors rather than the specific types of food consumed. However, it is important to further investigate this lack of relationship since, as previous research has shown, stress can significantly impact the type and quantity of food people choose [
68]. Many individuals report increased energy intake and alterations in their dietary preferences in response to stress, often favoring foods high in fat, sugar, calories, and palatability [
68]. In this regard, as observed in the case of emotional eating, cortisol plays a prominent role in mediating stress responses.
While the present research provides valuable insights into eating behavior, it is important to acknowledge certain limitations that should be addressed in future work. First, this study is cross-sectional, which restricts our ability to establish causal relationships between the variables studied. Additionally, it should be noted that the data were collected using self-report measures, an approach that is susceptible to potential inaccuracies stemming from memory bias or participants’ subjective perceptions. Furthermore, an uncontrollable factor in this study was the presence of clinical problems among the participants. It would be interesting to consider both a clinical population with eating disorders and a non-clinical population in future research. Despite these limitations, this study contributes valuable information that can help to analyze the relationships between the three constructs presented.