Version 1
: Received: 26 November 2023 / Approved: 27 November 2023 / Online: 27 November 2023 (09:09:47 CET)
How to cite:
FERRARI, I.V. Novel Perspectives on Amentoflavone's Influence on Cell Cycle and Apoptosis: Exploring Theoretical Modulation of Serine/Threonine-Kinases. Preprints2023, 2023111690. https://doi.org/10.20944/preprints202311.1690.v1
FERRARI, I.V. Novel Perspectives on Amentoflavone's Influence on Cell Cycle and Apoptosis: Exploring Theoretical Modulation of Serine/Threonine-Kinases. Preprints 2023, 2023111690. https://doi.org/10.20944/preprints202311.1690.v1
FERRARI, I.V. Novel Perspectives on Amentoflavone's Influence on Cell Cycle and Apoptosis: Exploring Theoretical Modulation of Serine/Threonine-Kinases. Preprints2023, 2023111690. https://doi.org/10.20944/preprints202311.1690.v1
APA Style
FERRARI, I.V. (2023). Novel Perspectives on Amentoflavone's Influence on Cell Cycle and Apoptosis: Exploring Theoretical Modulation of Serine/Threonine-Kinases. Preprints. https://doi.org/10.20944/preprints202311.1690.v1
Chicago/Turabian Style
FERRARI, I.V. 2023 "Novel Perspectives on Amentoflavone's Influence on Cell Cycle and Apoptosis: Exploring Theoretical Modulation of Serine/Threonine-Kinases" Preprints. https://doi.org/10.20944/preprints202311.1690.v1
Abstract
Docking results suggest that Amentoflavone has a higher affinity for investigated Serine/threonine-protein kinases, indicating potential interactions that may influence the activities of these kinases. The negative values of the binding energies signify strong and stable interactions, suggesting that Amentoflavone could be a key molecule in modulating the functions of these kinases, which play crucial roles in cellular processes such as cell cycle regulation and apoptosis.
Keywords
serine/threonine protein kinase; amentoflavone; molecular docking; apoptosis
Subject
Medicine and Pharmacology, Medicine and Pharmacology
Copyright:
This is an open access article distributed under the Creative Commons Attribution License which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
