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Mammal Aging as a Programmed Life Cycle Function—Resolving the Cause and Effect Conundrum
Version 1
: Received: 16 July 2023 / Approved: 18 July 2023 / Online: 19 July 2023 (04:18:37 CEST)
How to cite: Goldsmith, T. Mammal Aging as a Programmed Life Cycle Function—Resolving the Cause and Effect Conundrum. Preprints 2023, 2023071278. https://doi.org/10.20944/preprints202307.1278.v1 Goldsmith, T. Mammal Aging as a Programmed Life Cycle Function—Resolving the Cause and Effect Conundrum. Preprints 2023, 2023071278. https://doi.org/10.20944/preprints202307.1278.v1
Abstract
As recently as 2002 programmed aging in mammals was widely thought to be theoretically impossible based on generally accepted concepts regarding the evolution process. However, as described in this article, genetics discoveries, results of selective breeding, and other direct evidence strongly support the idea that aging creates an evolutionary advantage and that therefore complex biological mechanisms evolved that control mammal aging. Like similar life-cycle programs that control reproduction, growth, and menopause the aging program can adjust the aging trait during an individual’s life to compensate for temporary or local changes in external conditions that alter the optimum lifespan for a particular species population. In addition, genetics discoveries strongly support the evolvability concept to the effect that sexually reproducing species can evolve design features that increase their ability to evolve, and that aging is one such feature.
Keywords
evolution, evolvability, biology, gerontology, lifespan, senescence, genetics
Subject
Biology and Life Sciences, Aging
Copyright: This is an open access article distributed under the Creative Commons Attribution License which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
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